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Critical analysis and explanation of pathogenesis based on Mr. Farouq’s condition case study,
1. Critical analysis and explanation of pathogenesis of Mr. Farouq’s condition
As we can see in the patient’s condition, he was diagnosed with pneumonia and his condition was deteriorating due to pain in chest and high severity of cough. He was inflicted by bacteria or micro-organisms which is associated with risk factors such as cystic fibrois, asthma, diabetes and heart failures. However, as per the given condition, the patient was vulnerable to poor ability to cough thus he developed a weak immune system. His respiratory tract might be infected with virus that can develop pneumonia or it can be caused by airbourne droplets those are imhaled through mouth and nose probably. The lungs are also producing mucus, prevention of which can result in pneumonia. Cell death for Mr. Farouq is quite natural as invasion takes place through lungs. As the immune systems responds to the lungs infection, the damage and inflammation becomes severe. The reason for inflammation is white blood cell loss and affecting the mono-nuclear cells (Kropski et al. 2015). As this is a virus borne attack, along with the lungs, other body parts get affected through it. Finally, it makes the body organs vulnerable to bacteria attack.,
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There will be tow ways to manage the damage caused by pneumonia; the first one is vaccination and second one is medication. This must be taken into a controlled environment, where further chances of getting the virus affected is minimal. This strategy can be effective in terms of decreasing the risk of invasion of pneumococcal disease (Dickson, Erb-Downward & Huffnagle, 2014). However, for the patient, a daily dose of amantadine or rimantadine can be fruitful as they are helping in preventing deterioration of farouq’s condition. Yet, these applications are associated with side effect so zanamivir or oseltamivir can be another good choice for the patient. The benefit of using these medications is that there are no side effects as well decreasing the chances of developing symptoms if exposed to the risk factors somehow.
3. Critical analysis of arterial blood gas results
This test shows the amount of carbon dioxide in the blood along with acidity of the blood. The test reveals that carbon dioxide presence in his body is quite high that refers to abnormal results damage can occur to his lungs and kidneys. The higher range that normal value indicates breathing problem and lower oxygen value means the patient must be supplied with external air support. However, the pathogenesis also found problems in his breathing and lungs those need be cured before getting him into a good position again. His metabolic condition might be dislodged for affect in kidney. Overall, he must be supported with life saving medication systems and any side effect from the used medication can damage the immunes system to more extent. It also guides treatment plan and ventilator management in order to save the life of a person and reduces his chances of getting more affected by virus.
Reference list
Kropski, J. A., Pritchett, J. M., Zoz, D. F., Crossno, P. F., Markin, C., Garnett, E. T., ... & Choi, L. (2015). Extensive phenotyping of individuals at risk for familial interstitial pneumonia reveals clues to the pathogenesis of interstitial lung disease. American journal of respiratory and critical care medicine, 191(4), 417-426.
Dickson, R. P., Erb-Downward, J. R., & Huffnagle, G. B. (2014). Towards an ecology of the lung: new conceptual models of pulmonary microbiology and pneumonia pathogenesis. The lancet Respiratory medicine, 2(3), 238-246.
Feldman, C., & Anderson, R. (2015). Community-acquired pneumonia: pathogenesis of acute cardiac events and potential adjunctive therapies. Chest, 148(2), 523-532.
Dickson, R. P., Erb-Downward, J. R., & Huffnagle, G. B. (2014). Towards an ecology of the lung: new conceptual models of pulmonary microbiology and pneumonia pathogenesis. The lancet Respiratory medicine, 2(3), 238-246.
Feldman, C., & Anderson, R. (2015). Community-acquired pneumonia: pathogenesis of acute cardiac events and potential adjunctive therapies. Chest, 148(2), 523-532.
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